Category:We're too cool for a label

Figures 1, 3, and 4

Figures 2 and 3.

Figure 4. Direct sequencing of Crouzon syndrome FGFR2 mutations.

Figure 3, 4, 5, 6, 8.

dfxr Overexpression Lines and Nulls Display Postural and Locomotive Defects as Viable Adults; Overexpression and Loss-of-Function of dFXR Gave Rise to Structural and Functional Eye Phenotypes; dfxr Mutants and Overexpression Lines Differentially Affect Synaptic Growth and Structure; Figure 6. dfxr Nulls and Overexpression Lines Differently Affect Evoked and Spontaneous Neurotransmission; Synaptic Phenotypes of dfxr Nulls Are Suppressed by futsch Mutants

Table 2. Molecular alterations associated with torpedo mutations.

Figure 2. Defects in the inner and middle ear in Six1-deficient mice.

Figure 1. Postnatal Gonadal Sex Reversal in XX Foxl2 Conditional Mutant Mice

Figures 1, 2, and 3

Disruption of genes encoding Rri1p/Csn5p-associated proteins causes accumulation of Rub1p-modified Cdc53p.. figure 1

similar to fission yeast csn mutants, budding yeast Δcsn strains have a biochemical defect in the removal of Rub1p from Cdc53p, suggesting that the Rri1p/Csn5p, Csn9p, Rri2p/Csn10p, Pci8p/Csn11p, and Csi1p complex represents a budding yeast equivalent of the CSN complex described in fission yeast, plants, and human cells. figure2

Figures 4 and 5

FIG. 7. Western blot analysis of 1,25-(OH)2Ds receptor in

activated human lymphocytes.

figure 2 , figure 3

Figure 3

Figures 2, 3, 4, 5, and 6.

Figure 5. The myo2-66 mutation

disrupts actin cables. Proliferating homozygous myo2-66 (B, D, F) or wad-type (A, C, E) diploid cells.

Figure 1. Phenotype of the ms1 mutant.