Category:Team The Jackson 5

Figure 3 shows knock-out adcA mutant grew significantly less than wild-type adcA in zinc-containing media.

Figures 4 shows wild-type lmb grew and knock-out lmb didn't grow in media containing ZnCl2.

In Figure 3, the experiment shows that C3b is cleaved by the F1/FH complex which inhibits opsonization that is needed to induce phagocytosis.

Figure 2

Streptococcus pyogenes uses Factor H to degrade C3B which is responsible for opsinization. This results in evading phagocytosis.

Streptolysin o acts synergistically with NADase+ SPN to destroy cell membranes in infected cells and cause necrosis. In cells infected with mutant types of these proteins less then 10% of the cell membranes were destroyed.

NADase acts synergistically with SLO to cause cell death in all essentially all of the cells they infect. Mutant forms of the protein only caused cell death in less then 10% of the infected cells.

Figure 9. Charts B, C, and D indicate bacteria lacking PepO gene had significantly less adherence, thus reducing rates of cell invasion and bacteria survival.

Figure 2

Contributes to pathogenesis: associated with cell membrane fusion and attachment.

Table 2.

HasC1 aids in the production of a hyaluronic acid capsule. The capsule helps to evade phagocytosis.

Table 2.

UDG aids in the production of the hyaluronic acid capsule which mimics hair connective tissue.

Table 2.

HasA is involved in the production of the hyaluronic acid capsule. Hyaluronic acid mimics host connective tissue.

Table 2.

HasC2 aids in the production of a hyaluronic acid capsule. The capsule helps to evade phagocytosis.

Figure 2

Strep A species lack hasABC genes needed for hyaluronic acid capsule formation, which contributes to Strep species pathogenesis.

Figure 4

Analyzed various strains cytotoxic effects on keratinocytes.

Figure 2