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Figure two shows coimmunoprecipitation with dopamine receptor d1 in rat striatum.

Figure 2 shows that subunit 5HT3B is only expressed on the cell membrane when it is co-expressed with 5HT3A

Figure 5 illustrates a concentration-response curve where the percent of maximal response from the 5HT3C subunit increased when presented with agonists 5-HT and mCPBG and decreased when presented with antagonists azasetron and ondansetron.

Figure 5 illustrates a concentration-response curve that shows that the percent of maximal response from the 5HT3E subunit increased when presented with agonists 5-HT and mCPBG and decreased when presented with antagonists such as azasetron and ondansetron.

Figure 5 illustrates a concentration-response curve that shows that the percent of maximal response from the subunit increased when presented with agonists 5-HT and mCPBG and decreased when presented with antagonists such as azasetron and ondansetron.

Figure 3. MYBBP1A induces anoikis in normal breast and breast cancer cells through siRNA knockdown.

Based on figure 6 knockout mice (alpha-actin-3 deficient)show an increased amount of EDL muscle recovery.

Based on figure 4 and figure 5 activity of Lipoprotein Lipase (LPL) was enhanced by Apolipoprotein C-II (apoC-II)

Based of figure 7 and figure 8 wound healing of Calu-3 epithelial lung cells is enhanced by Interferon (IFN)-γ (Interferon gamma).

Figure 7 shows that Interleukin-4 (IL-4) inhibits the migration of the Calu-3 epithelial cells, but it does not directly prove the inhibition of wound healing.