Category:Team Mighty Morphin GO Annotators

In figure 6, the authors demonstrate that homozygous relaxin knockout mice displayed abnormal nipple development after parturition.

In table 1, the authors demonstrate that homozygous relaxin knockout mice displayed altered mammary gland morphogenesis.

In table 2, the authors demonstrate that homozygous relaxin knockout mice had more Bax and caspase-9 positive cells in the testis.

In figure 2 E,F,G &H, the authors demonstrate that there is a reduction in mature spermatozoa formation in seminiferous tubules of homozygous relaxin knockout mice.

In figure 3 A&C, the authors demonstrate that homozygous mutant male mice, deficient in INSL3, exhibited bilateral cryptorchid testes located high in the abdomen.

In figure 6, the authors demonstrate that porcine relaxin increases the proportion of cells that are positive for proliferating cell nuclear antigen, indicating cell proliferation.

In figure 6A, the authors demonstrate that the addition of human relaxin increases the activity of matrix metalloproteinase activity in human prostate tumor cells.

In figure 8, the authors demonstrate that tumors, comprised of human prostate tumor cells, had greater microvessel density when made to overexpress human relaxin.

In figure 4, the authors demonstrate that the neutralization of endogenous relaxin during late pregnancy has a negative effect on cervical epithelial and stromal cell proliferation.

In figure 6, the authors demonstrate that the neutralization of endogenous relaxin during late pregnancy increases apoptosis in cervical epithelial and stromal tissue.

In table 4, the authors demonstrate that the addition of porcine relaxin during in vitro culture increases the number of cells in porcine blastocysts.

In figure 1, the authors demonstrate that the addition of porcine relaxin increased the proportion of live boar spermatozoa that had undergone the acrosome reaction.

In figure 2, the authors show that porcine relaxin therapy in ovariectomized gilts restored collagen organization in the cervix during pregnancy.

In figure 1, the authors demonstrate that the addition of human relaxin stimulates the expression of pro-MMP-1 at both the mRNA and protein level.

The authors showed that treatment with PTHrP increased osteoclast proliferation. Figure 1.

In figure 2, the authors demonstrate that the addition of porcine relaxin increases the glucose import in boar spermatozoa.

In figure 1, the authors demonstrate that human RXFP1 has a positive influence on cAMP production in the presence of its ligand.

In figure 1, the authors demonstrate that the addition of porcine relaxin increases the motility of boar spermatozoa.

In table 2, the authors demonstrate that the addition of porcine relaxin during in vitro maturation reduced the the proportion of porcine oocytes that retained a germinal vesicle and increased the proportions of porcine oocytes that reach metaphase 2 (MII), both of which are characteristic of oocyte maturation.

In figure 7, the authors demonstrate the presence of porcine RXFP2 on the plasma membrane of porcine oocytes and embryos using immunofluoresence.