Category:Team Loony Toons

Figure 4 &5. Mutant TRAM2 inhibits collagen synthesis by hepatic stellate cells

Figure 2. (and 1)TRAM2 is expressed in activated haptic stellate cells

Figure 2 shows PrlA (SecY) is enriched in the plasma membrane by immunoblotting using anti-peptide antisera

Figure 4. NFB42 is cytoplasmic, showing high expression in dendrites in many parts of the brain.

Figure 5. Expression of protein inhibited cell growth

Figure 4. High expression of protein in brain neural axon

Figure 4. Majority of expression is seen in the cytoplasm.

Figure 3 and Figure 5. TBB2B mutants poorly produce competent heterodimers for microtubule polymerization.

Figure 2. Tubb2b expression was knocked-down by 50% in embryonic cells. This resulted in the arrest of neuron migration.

Figure 2. When tissues were treated with a permeability marker and different strains of cholera, the strains producing the toxin (inferred from fig 1) showed increased permeability in the tight junctions

Fig 3 shows mRNA is degraded by purified Phl p Vb

Fig 2. Absence of the labyrinth layer in the mutant phenotype. More specifically as seen in Fig.2 D-F there is no invagination of the allantois into the chorion, which is necessary for the formation of the labyrinth layer, in the mutants like there is in the wild type.

Figure 1c. Decrease in DNA synthesis when increase PTB expression.

Figure 2. Shows the crystal structure of native NX-5.

Table 2. Shows the ratios of isomerization products of NX-5 and mutants used to elucidate the product specificity role of the 10 residue loop adjacent to the catalytic site.

Figure 3 shows an immunoelectron microscope picture of epitope tagged protein that is located in the membrane where the C-terminus side is inside the cell.

Figure S4 of the supplementary data indicates nuclear localization of Snu66 in S. cerevisiae.

Figure 2 shows the AiiA gene product inactivation of autoinducers over time.