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Figure 1

Figure 2

Figure 3 shows the expression of HMGB1 in normal and SLE CD4+ T cells.

Figures 1B-E: The majority of changes to regulation of genes were dependent on type I IFN and not PRR stimulation. Thus type I IFN can be concluded to be the main variable for which maturation and metabolic shift to glycolysis after poly IC stimulation relies on.

Figure 3: LPS upregulation of mRNA expression of Nalp3 led to an increase in caspase, a protease known to play a role in inflammation.

Table 5: High HBV load led to the increase in AIM2 expression which in turn resulted in a positive correlation with caspase-1 and IL-1β observed.

Figure 1A: Unlike wild-type splenic cells, Aim2-null splenic cells had higher basal levels of STAT1, p202, p-STAT1, & viperin, an IFN-inducible protein.

Figure 4A shows that the protein is partially localized in the nucleus.

Figure 1B shows that CED-3 is a cysteine protease because when the mutant CED or an inhibitor was used, cleavage products were not present.