Category:Team Robots In Disguise

Fig 4

Cigarette smoke can lead to the over regulation of MiR-101. MiR-101 is one of the proteins that is responsible for regulating the CFTR protein chloride channels in the lungs. The CFTR chloride channel is responsible for carrying chloride ions across the membrane. Mutations of the chloride channel can cause mucous to accumulate on the membrane which is a sign of cystic fibrosis. Figure 6 shows that MiR-101 is up regulated in is COPD patients with histories of smoking. The up regulation in COPD patients with a history of smoking showed great suppression in the CFTR protein.

In figures 4A the effects MAP3K1 mutations on phosphorylation of downstream targets were examined by quantitative immunoblot analysis. Levels of phospho-ERK were increased in all the mutant cell lines in comparison to wild-type

In figure 4A shows a western blot which shows that after OA treatment, cells with Msn overexpression exhibited higher levels of T312 phosphorylation

In Figure 2 The effects of MINK and TNIK knockdown on neuronal morphology were visualized by co-transfected DsRed. Both MINK-RNAi and TNIK-RNAi significantly reduced arbor complexity relative to control neurons

In figure 2 the effects of MINK and TNIK knockdown on neuronal morphology were visualized by co-transfected DsRed.Both MINK-RNAi and TNIK-RNAi significantly reduced arbor complexity relative to control neurons

Figure 3 shows a western blott which shows that N and P were detected in fractions 11–15 and again in fractions 17–18 which indicates that most of N formed complexes with P which inhibits the binding of N to RNA