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|Status||Page||User||Date/Time||GO Term (Aspect)||Reference||Evidence||Notes||Links|
|BACSU:MCPC||Mcfadd48, MichSt14A 40||2014-04-06 17:55:15 CDT||GO:0050918 positive chemotaxis (P)||PMID:23038252||ECO:0000314 direct assay evidence used in manual assertion|
Table 1 shows the results from a capillary assay, indicating that McpC alone can support chemotaxis to 17 amino acids.
|ARATH:F4IU18||Mcfadd48, MichSt14A 40||2014-04-06 17:44:22 CDT||GO:0000302 response to reactive oxygen species (Figure 7 shows that the rol5-1 mutant is hypersensitive to hydrogen peroxide and hyposensitive to the ROS scavenger CuCl2, indicating the importance of ROL5 in the sensing of and response to ROS.)||PMID:20530756||IMP: Inferred from Mutant Phenotype||challenge|
|PSEAE:Q9HXI7||Mcfadd48, MichSt14A 40||2014-04-06 16:30:36 CDT||GO:0006979 response to oxidative stress (P)||PMID:24466226||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 5 shows decreased survival under oxidative stress conditions in an iscR mutant. Tn7-borne expression of iscR in the iscR mutant restored survival percentage to that of wild-type P. aeruginosa.
|AEDAE:Q534Q6||Mcfadd48, MichSt14A 40||2014-04-06 13:58:06 CDT||GO:0002218 activation of innate immune response (P)||PMID:15722339||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 4A shows transfection of REL1A alone can induce expression of Cecropin A and Defensin A in Aag2 cells. Figure 4D shows RNAi knockdown of REL1 decreases fungal infection survival in mosquitoes.
|XANOB:G7TJY8||Mcfadd48, MichSt14A 40||2014-04-06 13:13:00 CDT||GO:0009405 pathogenesis (P)||PMID:24675748||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 2A shows that a XOC_1550 mutant causes significantly shorter lesions, and Figure 2E shows that XOC_1550 mutant is impaired in its ability to grow in rice, identifying Xrp5 as a critical protein in pathogenesis
|DICD3:E0SG24||Mcfadd48, MichSt14A 40||2014-04-05 23:48:31 CDT||GO:0045892 negative regulation of transcription, DNA-templated (P)||PMID:22267675||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 2A shows that in a slyA mutant, hrp genes controlling the Type 3 Secretion System are overexpressed.
|ANOGA:B2FXG9||Mcfadd48, MichSt14A 40||2014-04-05 20:31:16 CDT||GO:0002218 activation of innate immune response (P)||PMID:16076953||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 3A shows that silencing of either domain of REL2 results in significant reduction of CEC1 (Cecropin A) expression.
|ENTFA:Q82ZP8||Mcfadd48, MichSt14A 40||2014-04-04 11:16:34 CDT||GO:0033554 cellular response to stress (P)||PMID:22092815||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 2 indicates that a slyA mutant strain is significantly impaired in its ability to grow under bile salt stress (in the presence of 0.08% bile salts), compared to wild-type E. faecalis
|ENTFA:Q82ZP8||Mcfadd48, MichSt14A 40||2014-04-04 11:16:34 CDT||GO:0033554 cellular response to stress (P)||PMID:22092815||ECO:0000314 direct assay evidence used in manual assertion|
Figure 1 shows induction of slyA under bile salt stress conditions.
|STRMU:Y1061||Mcfadd48, MichSt14A 40||2014-04-04 11:04:18 CDT||GO:0030695 GTPase regulator activity (F)||PMID:24659773||ECO:0000315 mutant phenotype evidence used in manual assertion|
Figure 4: An endpoint colorimetric assay indicated that the GTPase activity of Ffh alone, as well as that of Ffh/scRNA, was significantly increased upon addition of YlxM.
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Pages in category "MichSt14A 40"
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