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Cacao

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GO:0009231riboflavin biosynthetic processPMID:9473052IMP: Inferred from Mutant Phenotype P
This annotation made on page: BACSU:RIBC
By: Leferej2 (group Team Buckminster Fullerenes) on 2012-03-29 12:17:35 CDT.




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Entry TypeChallenging User,GroupTime/DateChallenge ReasonPoints/Assessment
ChallengeJenni162,
Team Gene-Yes
2012-04-05 20:59:52 CDT

Figure 4 shows FMN is a regulator of riboflavin production. The ribC mutant cannot produce FMN adenylyltransferase, therefore it cannot repress FMN and consequently riboflavin production is not repressed. This figure does not show that ribC necessarily changes riboflavin production because at low levels of FMN both the mutant and the wild type produced the same amount of riboflavin. This figure only shows FMN's effect on the gene. A more appropriate GO term is 3919 (FMN adenylyltransferase activity), because when Figure 3 and Figure 4 are both analyzed, it is shown that FMN affects riboflavin production, and also that a lack of FMN adenylyltransferase (as is the case in the ribC mutant) does not allow for the regulation of riboflavin production.

0
ChallengeJenni162,
Team Gene-Yes
2012-04-05 20:59:30 CDT

Figure 4 shows FMN is a regulator of riboflavin production. The ribC mutant cannot produce FMN adenylyltransferase, therefore it cannot repress FMN and consequently riboflavin production is not repressed. This figure does not show that ribC necessarily changes riboflavin production because at low levels of FMN both the mutant and the wild type produced the same amount of riboflavin. This figure only shows FMN's effect on the gene. A more appropriate GO term is 3919 (FMN adenylyltransferase activity), because when Figure 3 and Figure 4 are both analyzed, it is shown that FMN affects riboflavin production, and also that a lack of FMN adenylyltransferase (as is the case in the ribC mutant) does not allow for the regulation of riboflavin production.

8
Private
Assessment
Mkline2012-04-11 12:06:29 CDTYou need to be an instructor to view these notes.Requires Changes