Category:Team I broke mitosis

Figure 2c shows that in p53-/- cells there is a significant increase in lipid formation compared to p53+/+ cells with or without DHEA in humans

Organism: Human

Figure 1a shows that in p53-/- cells there is a significant increase in oxidative pentose phosphate pathway flux compared to p53+/+ cells in humans

Organism: Human

Figure 4C shows that " Gln3-myc13p runs faster in hsf1 R206S, F256S cells compared to HSF1 cells." The TOR kinase controls the phosphorylation of HSF1. Dephosphorylated proteins run faster than phosphorylated in yeast.

Figure 6 shows that PAX2 interacting with KDM1A (called LSD1 in paper) causes negitive regulation of neuronal differentiation in mice.

Figure 2D shows how KDMA1 (called LSD1 in paper) is associated with the NuRD complex in mice.

Figure 2C shows that PAX2 (Pax2 in paper) associates with the NuRD complex in mice.

Figure 2 shows Tmprss3 negatively regulates multiple proteins related to Kcnma1 as shown in green portion of Figure 2B,C.

Figure 5B shows that the inhibition of LSD1 (alpha-LSD1), had significantly higher levels of H3K4me1 (mono-methylation) versus the functioning LSD1, denoted as KDM1A in UNIPROT, in mice.

Figure 5B shows that the inhibition of LSD1 (alpha-LSD1), had significantly higher levels of H3K4me2 (di-methylation) versus the functioning LSD1 denoted as KDM1A in UNIPROT, in mice.

Figure 2 shows Tmprss3 positively regulates multiple proteins related to Kcnma1 as shown in red portion of Figure 2B,C.

Figure 2C shows protein import into mitochondrial matrix. Phosphorylation-mimicking OMG4 mutation (S568D) shows reduced % pFAD imported compared to OM64 WT and phospho-deficient OM64 mutation (S568A) in Arabidopsis Thaliana.

Figure 4 shows a significant decrease in mRNA production in the HSF1KO-HD in mice relative to the control group. This indicates that HSF1 is a positive regulator of gene expression in Nfatc2, Pdzk3, and Cryab in heart, spleen and straitium tissues.