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Gonzalez-Cabo, P, Ros, S and Palau, F (2010) Flavin adenine dinucleotide rescues the phenotype of frataxin deficiency. PLoS ONE 5:e8872
Friedreich ataxia is a neurodegenerative disease caused by the lack of frataxin, a mitochondrial protein. We previously demonstrated that frataxin interacts with complex II subunits of the electronic transport chain (ETC) and putative electronic transfer flavoproteins, suggesting that frataxin could participate in the oxidative phosphorylation.
Adenosine Triphosphate/biosynthesis; Animals; Caenorhabditis elegans/genetics; Caenorhabditis elegans/physiology; Dose-Response Relationship, Drug; Flavin-Adenine Dinucleotide/pharmacology; Gene Knockdown Techniques; Iron-Binding Proteins/genetics; Models, Biological; Oxidative Phosphorylation; Phenotype; Recombination, Genetic; Saccharomyces cerevisiae/genetics
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