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PMID:21949834
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| Citation |
Mouillesseaux, K and Chen, JN (2011) Mutation in utp15 disrupts vascular patterning in a p53-dependent manner in zebrafish embryos. PLoS ONE 6:e25013 |
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| Abstract |
Angiogenesis is the process by which the highly branched and functional vasculature arises from the major vessels, providing developing tissues with nutrients, oxygen, and removing metabolic waste. During embryogenesis, vascular patterning is dependent on a tightly regulated balance between pro- and anti-angiogenic signals, and failure of angiogenesis leads to embryonic lethality. Using the zebrafish as a model organism, we sought to identify genes that influence normal vascular patterning. |
| Links |
PubMed PMC3176792 Online version:10.1371/journal.pone.0025013 |
| Keywords |
Animals; Animals, Genetically Modified; Apoptosis; Blood Vessels/embryology; Blood Vessels/metabolism; Cloning, Molecular; Embryo, Nonmammalian/cytology; Embryo, Nonmammalian/metabolism; Embryonic Development; Gene Expression Regulation, Developmental; In Situ Hybridization; Mutation/genetics; Neovascularization, Physiologic; Phenotype; RNA, Messenger/genetics; Real-Time Polymerase Chain Reaction; Signal Transduction; Tumor Suppressor Protein p53/genetics; Tumor Suppressor Protein p53/metabolism; Zebrafish/embryology; Zebrafish/genetics; Zebrafish/metabolism; Zebrafish Proteins/physiology |
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