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PMID:15876356
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| Citation |
Sagane, K, Hayakawa, K, Kai, J, Hirohashi, T, Takahashi, E, Miyamoto, N, Ino, M, Oki, T, Yamazaki, K and Nagasu, T (2005) Ataxia and peripheral nerve hypomyelination in ADAM22-deficient mice. BMC Neurosci 6:33 |
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| Abstract |
ADAM22 is a member of the ADAM gene family, but the fact that it is expressed only in the nervous systems makes it unique. ADAM22's sequence similarity to other ADAMs suggests it to be an integrin binder and thus to have a role in cell-cell or cell-matrix interactions. To elucidate the physiological functions of ADAM22, we employed gene targeting to generate ADAM22 knockout mice. |
| Links |
PubMed PMC1142324 Online version:10.1186/1471-2202-6-33 |
| Keywords |
ADAM Proteins/deficiency; ADAM Proteins/genetics; ADAM Proteins/physiology; Animals; Ataxia/genetics; Ataxia/metabolism; Ataxia/pathology; Cells, Cultured; Female; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Nerve Fibers, Myelinated/metabolism; Nerve Fibers, Myelinated/pathology; Nerve Tissue Proteins/deficiency; Nerve Tissue Proteins/genetics; Nerve Tissue Proteins/physiology; Peripheral Nerves/metabolism; Peripheral Nerves/pathology; Peripheral Nervous System Diseases/genetics; Peripheral Nervous System Diseases/metabolism; Peripheral Nervous System Diseases/pathology |
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