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PMID:9802272

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Citation

Menegon, A, Board, PG, Blackburn, AC, Mellick, GD and Le Couteur, DG (1998) Parkinson's disease, pesticides, and glutathione transferase polymorphisms. Lancet 352:1344-6

Abstract

Parkinson's disease is thought to be secondary to the presence of neurotoxins, and pesticides have been implicated as possible causative agents. Glutathione transferases (GST) metabolise xenobiotics, including pesticides. Therefore, we investigated the role of GST polymorphisms in the pathogenesis of idiopathic Parkinson's disease.

Links

PubMed

Keywords

Aged; Australia; Blood-Brain Barrier/drug effects; Case-Control Studies; Environmental Exposure/adverse effects; Female; Genotype; Glutathione Transferase/classification; Glutathione Transferase/genetics; Humans; Logistic Models; Male; Middle Aged; Parkinson Disease, Secondary/chemically induced; Parkinson Disease, Secondary/genetics; Pesticides/adverse effects; Polymorphism, Genetic; Questionnaires

Significance

Annotations

Gene product Qualifier GO Term Evidence Code with/from Aspect Extension Notes Status

HUMAN:GSTP1

involved_in

GO:0007417: central nervous system development

ECO:0000304: author statement supported by traceable reference used in manual assertion

P

Seeded From UniProt

complete

See also

References

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