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PMID:177406

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Citation

Roehl, RA and Vinopal, RT (1976) Lack of glucose phosphotransferase function in phosphofructokinase mutants of Escherichia coli. J. Bacteriol. 126:852-60

Abstract

Phosphofructokinase (pfkA) mutants of Escherichia coli are impaired in growth on all carbon sources entering glycolysis at or above the level of fructose 6-phosphate (nonpermissive carbon sources), but growth is particularly slow on sugars, such as glucose, which are normally transported and phosphorylated by the phosphoenolpyruvate, (PEP)-dependent phosphotransferase system (PTS).

Links

PubMed PMC233222

Keywords

Escherichia coli/enzymology; Escherichia coli/growth & development; Escherichia coli/metabolism; Galactose/metabolism; Glucokinase/metabolism; Glucose/metabolism; Mannose/metabolism; Mutation; Phenotype; Phosphoenolpyruvate/metabolism; Phosphofructokinase-1/biosynthesis; Phosphotransferases/metabolism

Significance

Annotations

Gene product Qualifier GO Term Evidence Code with/from Aspect Extension Notes Status

ECOLI:PFKA

acts_upstream_of_or_within

GO:0006096: glycolytic process

ECO:0000315: mutant phenotype evidence used in manual assertion

P

Seeded From UniProt

complete

ECOLI:K6PF1

involved_in

GO:0006096: glycolytic process

ECO:0000315: mutant phenotype evidence used in manual assertion

P

Seeded From UniProt

complete


See also

References

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