Category:Team Protein Pushers

Figure 3. Decreased viral infection when IFITM3 present in chicken cells. Figure 4 and 5 also.

The FOX assay results in Figure 4 demonstrates that Ohr metabolizes hydrogen peroxide.

Figure 8: Western blots of cells transfected with WT VP40 and several hydrophobic mutants demonstrate that C-terminal domain plasma membrane penetration is critical for viral egress.

Figure 3 shows protein localization to the plasma membrane via immunofluresence assay

Syncytia only seen in mouse-human hybrid (IL-115) cells when cotransfected with CDV-HA (F protein source not as important, but also required for infection)

Figure 8 shows the hydrophobicity of mature AfpA proteins.

Figure 8 depicts the inhibition of endosome vesicle fusion by HP16 E5 protein

Figure 3 shows localization to the plasma membrane via immunofluorescence data

Figure 3: Acts as a repressor of INF-beta gene expression

Figure 3: RD cells transfected with Hsp90-alpha and Hsp90-beta specific siRNA. Only Hsp90-beta siRNA prevented EV71 capsid protein expression showing Hsp90-beta is involved in EV71 infection

Figure 2 shows the predicted amino acid sequence of HSP70 from goat to HSP 70 of cattle

Table 2 demonstrates the ability of the mutant strain without the cpdP gene to grow on various media. The inability of the mutant strain to grow on cAMP or cGMP demonstrates the function of the cpdP gene in cAMP metabolism.

Figure 1 shows the amino acid sequence alignment between SSB of Neisseria gonorrhoeae and SSB in E.coli

Figure 5 measures the amount of ATP hydrolyzed by RecA over time during strand exchange

Figure 1 shows an amino acid sequence alignment of recA from N. gonorrhoeae aligned to E.coli

Figure 3

Figure 2B, 2C show that when certain key amino acids are mutated (L213A, L295A, V298A) they do not induce the same amount of surface pressure into the plasma membrane as compared to WT

Figure 2. MgtC mutants showed decreased survival in human cells as compared to the WT

Figure 6

Figure 10 shows that APN-1 has the ability to repair various DNA lesions

Figure 10C shows that APN-1 exhibits a 3' repair diesterase activity

Figure 8

Figure 1 shows C.elegans APN-1 shows homology to Yeast Apn1, which is involved in DNA repair