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9VIRU:Q5ILC6
Contents
| Species (Taxon ID) | Bacillus phage GIL16c. (307240) | |
| Gene Name(s) | No Information Provided. | |
| Protein Name(s) | Putative LexA-like repressor (ECO:0000313 with EMBL:AAW33570.1) | |
| External Links | ||
| UniProt | Q5ILC6 | |
| EMBL | AY701338 | |
| RefSeq | YP_224104.1 | |
| ProteinModelPortal | Q5ILC6 | |
| GeneID | 4525346 | |
| KEGG | vg:4525346 | |
| KO | K01356 | |
| Proteomes | UP000001470 | |
| GO | GO:0004252 | |
| Gene3D | 1.10.10.10 | |
| InterPro | IPR006199 IPR011991 | |
| Pfam | PF01726 | |
| SUPFAM | SSF46785 | |
Annotations
| Qualifier | GO ID | GO term name | Reference | ECO ID | ECO term name | with/from | Aspect | Extension | Notes | Status |
|---|---|---|---|---|---|---|---|---|---|---|
| GO:0003700 |
transcription factor activity, sequence-specific DNA binding |
ECO:0000315 |
F |
In mutant SOS-deficient LexA hosts, transcription of the P1-P2-lacZ fusion gene, which is otherwise induced during SOS conditions, was not activated. Thus, LexA regulates transcription. Mutations in SOS box dinbox1 (a region on DNA), known to be important for LexA binding, makes it so that SOS can no longer be induced (Fig 1B), showing that LexA, which causes SOS induction, binds to dinbox1. |
complete | |||||
|
enables |
GO:0004252 |
serine-type endopeptidase activity |
ECO:0000256 |
match to sequence model evidence used in automatic assertion |
F |
Seeded From UniProt |
complete | |||
|
involved_in |
GO:0006508 |
proteolysis |
ECO:0000256 |
match to sequence model evidence used in automatic assertion |
P |
Seeded From UniProt |
complete | |||
Notes
References
See Help:References for how to manage references in GONUTS.
- ↑ Fornelos, N et al. (2015) Bacteriophage GIL01 gp7 interacts with host LexA repressor to enhance DNA binding and inhibit RecA-mediated auto-cleavage. Nucleic Acids Res. 43 7315-29 PubMed GONUTS page
