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The ACE I and ACTN3 X alleles determine speed and power for Lithuanian athletes. Table 3 shows that Lithuanian athletes who are carriers

of the ACE I/I and I/D as well as ACTN3 X/X and R/X genotypes have the potential to achieve better results in power-requiring sports

Figure 2 shows that the wet weight ratio(size) of the gastrocnemius muscle is at its lowest when the circulating myostatin levels are at their highest, in terms of days after injury.

Findings show follistatin expression increased during recovery phase of post-denervated muscle atrophy. Follistatin/Myostatin ratio can be used to track muscle recovery post nerve injury. Figure 3.

Figure 2. Figure 3. Shows SNP14 in CSN1S1 results in increased milk production.

Figure 2. Figure 3. Shows SNP14 of CSN1S1 results in decreased protein % in milk.

Figure 2. Figure 3. Shows SNP14 of CSN1S1 results in decreased fat % in milk.

Table 1 and Figure 2 show the Gastrocnemius, Quadriceps, and Triceps muscles all were significantly larger in weight(g) in myostatin deficient mice compared to normal myostatin mice.

The ACE I and ACTN3 X alleles determine speed and power for Lithuanian athletes. Table 3 shows that Lithuanian athletes who are carriers of the ACE I/I and I/D as well as ACTN3 X/X and R/X genotypes have the potential to achieve better results in power-requiring sports

Table 2 shows male and female athletes with the ACE I/I genotype had higher handgrip strength and

STEMP compared with male and female athletes having the ACE D/D genotype.

Table 2 shows Mstn–/– mice had higher rates of total and resting O2 consumption compared with Mstn+/+ mice, which is expected given the higher body weights of Mstn–/– mice. If the data are expressed as a function of body weight, however, Mstn–/– mice actually had lower rates of total and resting O2 consumption compared with Mstn+/+ mice.

Figure 4 shows mg fatty acids per g of muscle tissue is significantly less in myostatin depleted mice fed a high-fat diet.

Figure 3 shows blood glucose concentration is significantly lower in myostatin depleted mice fed a high-fat diet for 5 months, 90 min after ip glucose injection.

Figure 5 shows that myostatin depleted mice have a significantly lower incidence and severity of hepatic statosis when fed a high-fat diet for 5 months.

Figure 1a shows the increased mass of triceps muscle in Mstn -/- mice at different ages as well as an apparent dose-dependency on muscle mass.

Figure 1b-g shows the significant differences in fat pad mass between Mstn -/- and Mstn +/+ mice after 2 months of age.

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Fig. 2

Fig. 4

Fig. 4

Fig. 4 and Fig. 5

Fig. 4c and Fig.5b,5c

Fig. 3

Fig. 1

Fig. 5

Fig. 5c

Fig. 3

Fig. 7. Results were most notable in MD exposure. Negative regulator of flowering.

Figure 3

Figure 4

Figure 2

Figure 3

Figure 4, Figure 5

Figure 3,4,6

Figure 4

Fig. 1,2,3

Fig. 7

Fig. 3

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Fig. 6

Fig. 6

Fig. 2 shows that the SWI5-SFR1 complex functions in stabilization of the Rad51 filament

Fig. 2 shows the SWI5-SFR1 complex functions in stabilizing the Rad51 filament