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SALT1:D0ZMU5
Contents |
| Species (Taxon ID) | Salmonella typhimurium (strain 14028s / SGSC 2262). ([1]) | |
| Gene Name(s) | gidA (synonyms: mnmG) | |
| Protein Name(s) | tRNA uridine 5-carboxymethylaminomethyl modification enzyme MnmG
Glucose-inhibited division protein A | |
| External Links | ||
| EMBL | CP001363 | |
| ProteinModelPortal | D0ZMU5 | |
| SMR | D0ZMU5 | |
| PRIDE | D0ZMU5 | |
| GenomeReviews | CP001363_GR | |
| GO | GO:0005737 GO:0050660 GO:0002098 | |
| HAMAP | MF_00129 | |
| InterPro | IPR004416 IPR002218 IPR020595 | |
| Pfam | PF01134 | |
| TIGRFAMs | TIGR00136 | |
| PROSITE | PS01280 PS01281 | |
Annotations
| Qualifier | GO ID | GO term name | Reference | Evidence Code | with/from | Aspect | Notes | Status |
|---|---|---|---|---|---|---|---|---|
| GO:0002098 |
tRNA wobble uridine modification |
IEA: Inferred from Electronic Annotation |
P |
Seeded From UniProt |
||||
| GO:0005737 |
cytoplasm |
IEA: Inferred from Electronic Annotation |
C |
Seeded From UniProt |
||||
| GO:0005737 |
cytoplasm |
IEA: Inferred from Electronic Annotation |
C |
Seeded From UniProt |
||||
| GO:0005737 |
cytoplasm |
IEA: Inferred from Electronic Annotation |
SP_SL:SL-0086 |
C |
Seeded From UniProt |
|||
| GO:0008033 |
tRNA processing |
IEA: Inferred from Electronic Annotation |
P |
Seeded From UniProt |
||||
| GO:0008033 |
tRNA processing |
IEA: Inferred from Electronic Annotation |
P |
Seeded From UniProt |
||||
| GO:0008033 |
tRNA processing |
IEA: Inferred from Electronic Annotation |
P |
Seeded From UniProt |
||||
| GO:0050660 |
flavin adenine dinucleotide binding |
IEA: Inferred from Electronic Annotation |
F |
Seeded From UniProt |
||||
| GO:0050660 |
flavin adenine dinucleotide binding |
IEA: Inferred from Electronic Annotation |
F |
Seeded From UniProt |
||||
| GO:0051301 |
cell division |
IMP: Inferred from Mutant Phenotype |
P |
Figure 1 shows the mutant phenotype is incapable of proper cell division |
complete | |||
| GO:0007059 |
chromosome segregation |
IMP: Inferred from Mutant Phenotype |
P |
Figure 2 and 3 show that the gidA mutant phenotype has a defect in chromosome segregation causing filamentous morphology. |
complete | |||
| edit table |
Notes
References
See Help:References for how to manage references in GONUTS.
- ↑ 1.0 1.1 Shippy DC et al. (2012) Deletion of glucose-inhibited division (gidA) gene alters the morphological and replication characteristics of Salmonella enterica Serovar typhimurium. Arch Microbiol 194: 405-12 PubMed GONUTS page
